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The Molecular Basis of Cellular Defence Mechanisms

Welcome to Loot. Checkout Your Cart Price. Description Details Customer Reviews The field of lymphokine research has grown in parallel to the exciting developments around the two sets of cells which defend the body. While lymphokines are the "property" of immunologists, the molecular regulators of hemopoiesis CSFs belong to the hematologists. This book offers the rare opportunity to examine these separate fields of expertise together.

Review This Product No reviews yet - be the first to create one! Need help? Partners MySchool Discovery. Subscribe to our newsletter Some error text Name. Email address subscribed successfully. Pharmacol Ther.

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Energy turnover of vascular endothelial cells. Am J Physiol. Fatty acids are not an important fuel for coronary microvascular endothelial cells. Mol Cell Biochem. Energetic response of coronary endothelial cells to hypoxia. Mitochondrial metabolism of reactive oxygen species.

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Biochemistry Mosc. Mitochondrial formation of reactive oxygen species. J Physiol. Reactive oxygen and targeted antioxidant administration in endothelial cell mitochondria. Redox regulation of cardiac calcium channels and transporters. Cardiovasc Res. J Cell Sci. Effects of NO on mitochondrial function in cardiomyocytes: pathophysiological relevance.

Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide. Models of the diffusional spread of nitric oxide: implications for neural nitric oxide signalling and its pharmacological properties. Role of endothelium-derived nitric oxide in the regulation of cardiac oxygen metabolism: implications in health and disease. Circ Res.

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Does nitric oxide modulate mitochondrial energy generation and apoptosis. Nat Rev Mol Cell Biol. On the mechanism by which vascular endothelial cells regulate their oxygen consumption. Competitive, reversible, physiological? Inhibition of mitochondrial cytochrome oxidase by nitric oxide.

Nitric oxide and cardiac function: ten years after, and continuing. Modulation of cardiac contraction, relaxation and rate by the endothelial nitric oxide synthase eNOS : lessons from genetically modified mice. Nitric oxide and excitation-contraction coupling. J Mol Cell Cardiol. Biochem Biophys Res Commun. Role of calcium ions in regulation of mammalian intramitochondrial metabolism.

A role for calcium influx in the regulation of mitochondrial calcium in endothelial cells. Am J Physiol Cell Physiol. Mitochondrial calcium uptake stimulates nitric oxide production in mitochondria of bovine vascular endothelial cells.

Cell Calcium. Sci STKE. Intracellular sodium modulates mitochondrial calcium signaling in vascular endothelial cells. Mitochondria are excitable organelles capable of generating and conveying electrical and calcium signals. Putting out the fire: what terminates calcium-induced calcium release in cardiac muscle. Propagation of the apoptotic signal by mitochondrial waves. EMBO J. The mitochondrial permeability transition pore and its role in cell death.

my.kovalev.com.ua/modules/50-acheter-azithromycin.php Biochem J. Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death.

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Properties of the permeability transition pore in mitochondria devoid of Cyclophilin D. Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death. Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia. Biochim Biophys Acta. Transient mitochondrial permeability transition pore opening mediates preconditioning-induced protection.

Mitochondrial permeability transition pore opening during myocardial reperfusion—a target for cardioprotection. Fluctuations in mitochondrial membrane potential caused by repetitive gating of the permeability transition pore. Laser-stimulated fluorescence of submicrometer regions within single mitochondria of rhodamine-treated myocardial cells in culture. Reactive oxygen species ROS -induced ROS release: a new phenomenon accompanying induction of the mitochondrial permeability transition in cardiac myocytes. J Exp Med. Characterization of depolarization and repolarization phases of mitochondrial membrane potential fluctuations induced by tetramethylrhodamine methyl ester photoactivation.

FEBS J. Transient mitochondrial depolarizations reflect focal sarcoplasmic reticular calcium release in single rat cardiomyocytes. J Cell Biol. A peptide trivalent arsenical inhibits tumor angiogenesis by perturbing mitochondrial function in angiogenic endothelial cells. Cancer Cell.

Hypoxic pulmonary vasoconstriction: redox events in oxygen sensing.